A younger organism would naturally release more cytokines than and elderly one.

年轻的有机体自然会比年老的有机体放更多的细胞因子。

IL-5, on the other hand, activates eosinophils which promote an immune response by releasing more cytokines and leukotrienes.

另方面，IL-5激活嗜酸性粒细胞，放更多的细胞因子和白三烯来促进免疫反应。

If it hijacks the immune system, and a cytokine storm occurs?

如果被它操控了整个免疫系统 并且发生了细胞因子风暴？

Cytokines are thought to also enhances the activity of the NMDA receptors.

这细胞因子可能使NMDA受体的活性增强。

The release of cytokines causes symptoms like fever, drenching night sweats, and weight loss.

细胞因子的放会导致发、夜间盗汗和体重减轻等症状。

It works as an anti-inflammatory and similar to other remedies, influences cytokine production in the body.

它可以作为种抗炎药，和其他药物样，能够影响体内细胞因子的产生。

Cytokines are proteins released by cells to interact with each other and to trigger specific processes.

细胞因子是细胞间相互作用并触发特定程而放的蛋白质。

These helper T-cells produce small signaling molecules called cytokines, which activate B-cells and macrophages.

辅助T细胞会产生的信号分子，即细胞因子，激活B细胞和巨噬细胞。

Known for its antibacterial activity, it increases the release of inflammatory cytokines, which signal cells to repair themselves.

众所周知，它具有抗菌活性，能增加炎症细胞因子的放，而炎症细胞因子是细胞自我修复的信号。

Dr Robert Snelgrove from Imperial College London, among others, has theorised the notion of the so-called 'cytokine storm'.

伦敦帝国理工学院的罗伯特·斯内尔格罗夫博士等人提出了所谓“细胞因子风暴”理论。

Along with an increase in proteins called cytokines—which the researchers say might suggest a boost in immune activity.

同时种名为细胞因子的蛋白质含量升高，对此研究人员表示这可能表明免疫活性提升。

In response, the injured cells release inflammatory molecules called cytokines that activate the immune system to repair the injury.

对此反应，损伤的细胞放出 称为“细胞因子”的发炎分子用来激活免疫系统和修复损伤。

They cytokines also attract B-cells and macrophages as part of the inflammatory reaction.

细胞因子也会趋化B淋巴细胞和巨噬细胞，这也是炎症反应的部分。

They communicate mostly via tiny information proteins called cytokines.

它们主要称为细胞因子的微信息蛋白进行讯。

Give it just enough blood to stop the cytokines, but not so much that he bleeds to death.

给它足够的血液来阻止细胞因子的产生，但不要太多，以免他失血多而死。

If the T cell gets activated, it kicks into action, producing cytokines which helps to get other immune cells involved.

T细胞在被激活后，开始合成分泌细胞因子，进而调动其他更多的免疫细胞。

This leads to the flood of enzymes and degradation seen in osteoarthritis, but is driven by different inflammatory proteins called cytokines.

也出现骨关节炎中产生大量酶和削弱关节的现象，但由不同的炎症蛋白引起，这种蛋白称为细胞因子。

In chronic disease states, chemical messengers called cytokines mediate this pathologic process in the kidney, immune system, and the GI tract.

在慢性疾病中，称为细胞因子的化学信使在肾脏、免疫系统和胃肠道中介导了这病理程。

In rats and people, intentionally activating the immune system and triggering the release of cytokines increases anxiety.

在老鼠和人身上， 有意激活免疫系统并触发细胞因子的放会增加焦虑。

And what we find is that inflammatory molecules such as cytokines are increased in response to the diet.

我们发现，炎症分子如细胞因子会随着饮食而增加。